Inductive Camrelizumab and Apatinib for Patients With Locally Advanced and Resectable Oral Squamous Cell Carcinoma
- Sponsor
- Shanghai Jiao Tong University School of Medicine
- Study ID
- NCT04393506
- Phase
- PHASE1
- Status
- Completed
Conditions
- Inductive Therapy
- Oral Cancer
- Programmed Cell Death 1 Inhibitor
- VEGFR2 Inhibitor
Eligibility Criteria
- Sex
- ALL
- Age
- 18 Years - 75 Years
- Healthy Volunteers
- Not accepted
Interventions
- Camrelizumab — DRUGInductive therapy with Camrelizumab of 200mg, iv, qd, on day 1, 15, 29.
- Apatinib — DRUGInductive therapy with Apatinib of 250mg, po, qd, initiating on day 1, ending on the fifth day before surgery.
- Radical surgery — PROCEDURERadical surgery will be performed on the 42th-45th after initiation of inductive therapy
- Post-operative radiotherapy/chemoradiotherapy — RADIATIONPost-operative radiotherapy/chemoradiotherapy will be performed within 1.5 months after radical surgery, depending on the post-operative pathologic diagnosis.
Study Details
In patients with locally advanced oral squamous cell carcinoma (OSCC), due to the large tumor burden and neck lymph node metastasis, comprehensive treatment is recommended, including surgery, radiotherapy, chemotherapy and others. Pre-operative inductive therapy can reduce tumor volume, increase organ retention rate, and reduce distant metastasis rate. Vascular endothelial growth factor (VEGF) receptor in head and neck squamous cell carcinoma is over-expressed and associated with disease invasion and poor prognosis. The use of targeted therapy against VEGF can not only inhibit tumor neovascularization, but also make the effectiveness of chemotherapeutic agents. VEGF and VEGFR are closely related to immune escape. Tumor growth requires new blood vessels to supply nutrients and oxygen, and VEGF can stimulate neovascularization. However, tumor neovascularization is often abnormal and distorted, which prevents immune active substances from reaching the tumor site. After tumor hypoxia, high expression of VEGF will induce tumor cells to express programmed cell death protein-1 (PD-1), which further leads to immune escape. Targeted drugs against angiogenesis can relieve immunosuppression to a certain extent, and theoretically have a synergistic effect with anti-PD-1 immunotherapy. The innovation of this study is the combination of immune checkpoint inhibitor, Camrelizumab, and targeted drug against VEGFR, Apatinib, as an inductive therapy to treat the patients with locally advanced OSCC, followed with radical surgery and post-operative radiotherapy/chemoradiotherapy, the major pathologic response and safety will be evaluated as the primary surrogate endpoints, the 2-year survival rate and local recurrence rate will be the second endpoints.
Key Dates
- Start date
- Apr 23, 2020
- Status verified
- Nov 2023
- Primary completion
- Nov 2, 2020
- Completion
- Nov 10, 2023
Study Design
- Enrollment
- 21 participants (actual)
- Allocation
- NA
- Intervention model
- SEQUENTIAL
- Primary purpose
- TREATMENT
Arms
- Other: Inductive therapyInductive therapy with Camrelizumab and Apatinib, followed by radical surgery and post-operative radiotherapy/chemoradiotherapy.
Primary Outcome Measure
Major pathologic response [ Time Frame: One year ]
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