Combination of BTK Inhibitor Overcomes Drug-resistance in Refractory/Relapsed FLT3 Mutant AML
- Sponsor
- Nanfang Hospital, Southern Medical University
- Study ID
- NCT03642236
- Phase
- PHASE2/PHASE3
- Status
- Unknown
Conditions
- Acute Myeloid Leukemia
- Brutons Tyrosine Kinase
- FLT3-ITD Mutation
Eligibility Criteria
- Sex
- ALL
- Age
- 14 Years - 60 Years
- Healthy Volunteers
- Not accepted
Interventions
- Ibrutinib — DRUGIbrutinib 420mg day -3 to d14 combining with chemotherapy with/without sorafenib based on whether the patients are naive to sorafenib before relapse.
Study Details
Clinical efficacy of FLT3 inhibitors combining with chemotherapy is usually transient and followed by emergence of drug-resistance in FLT3-ITD mutant AML. BTK is reported to be a therapeutic target in this subtype leukemia. Our previous study showed inhibition of BTK onvercome drug-resistance to FLT3 inhibitors/chemotherapy in refractory/relapsed FLT3 mutant AML. In this prospective randomized controlled study, the efficacy and safety of combination of BTK inhibitor with chemotherapy with/without FLT3 inhibitor in refractory/relapsed FLT3 mutant AML are evaluated.
Key Dates
- Start date
- Aug 31, 2018
- Status verified
- Aug 2018
- Primary completion
- Aug 31, 2022
- Completion
- Sep 30, 2023
Study Design
- Enrollment
- 122 participants (estimated)
- Allocation
- RANDOMIZED
- Intervention model
- PARALLEL
- Primary purpose
- TREATMENT
Arms
- Experimental: BTK treatmentIbrutinib 420mg day -3 to d14; Decitabine 20mg/m2 d1-5; Aclacinomycin 10mg/m2 d1-5; Cytarabine 15mg/m2 q12h d1-14; G-CSF 200ug/m2 -12h to d14; Sorafenib 0.4g bid continously at the condition of being naive to sorafenib.
- Active Comparator: BTK-free treatmentDecitabine 20mg/m2 d1-5; Aclacinomycin 10mg/m2 d1-5; Cytarabine 15mg/m2 q12h d1-14; G-CSF 200ug/m2 -12h to d14; Sorafenib 0.4g bid continously at the condition of being naive to sorafenib.
Primary Outcome Measure
CR rate [ Time Frame: After the first and second cycle induction ]
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