CBP501 Evidence: Trial Results and Peer-Reviewed Publications

Hipa.ai Research · Source: PubMed & ClinicalTrials.gov / AACT · Last updated:

The clinical evidence base for CBP501 comprises 8 peer-reviewed publications across 6 journals, 0 pivotal-trial primary-outcome rows reported to ClinicalTrials.gov, spanning indications including Pancreatic Neoplasms, Neoplasms, Mesothelioma, Malignant, and Lung Neoplasms. Most recent publication: A multicenter, randomized phase 2 study to establish combinations of CBP501, cisplatin and nivolumab for ≥3rd-line treatment of patients with advanced pancreatic adenocarcinoma., Eur J Cancer, 2024.

Top peer-reviewed publications

Curated set of pivotal-trial result papers and recent publications in high-tier journals.

  1. Phase I studies of CBP501, a G2 checkpoint abrogator, as monotherapy and in combination with cisplatin in patients with advanced solid tumors.
    Shapiro GI, Tibes R, Gordon MS, et al. · Clin Cancer Res · 2011
  2. A multicenter, randomized phase 2 study to establish combinations of CBP501, cisplatin and nivolumab for ≥3rd-line treatment of patients with advanced pancreatic adenocarcinoma.
    Enzler T, Nguyen A, Misleh J, et al. · Eur J Cancer · 2024
    PubMed: PMID 38422585 · NCT04953962 · Pancreatic Neoplasms
  3. Sensitization of cancer cells to DNA damage-induced cell death by specific cell cycle G2 checkpoint abrogation.
    Suganuma M, Kawabe T, Hori H, et al. · Cancer Res · 2000
    PubMed: PMID 10606229 · NCT00700336 · Mesothelioma, Malignant
  4. Cell cycle phenotype-based optimization of G2-abrogating peptides yields CBP501 with a unique mechanism of action at the G2 checkpoint.
    Sha SK, Sato T, Kobayashi H, et al. · Mol Cancer Ther · 2007
  5. CBP501-calmodulin binding contributes to sensitizing tumor cells to cisplatin and bleomycin.
    Mine N, Yamamoto S, Saito N, et al. · Mol Cancer Ther · 2012
    PubMed: PMID 21831962 · NCT04953962 · Pancreatic Neoplasms
  6. Activation of Nrf2 pathways correlates with resistance of NSCLC cell lines to CBP501 in vitro.
    Mine N, Yamamoto S, Kufe DW, et al. · Mol Cancer Ther · 2015
  7. Screening of a library of T7 phage-displayed peptides identifies alphaC helix in 14-3-3 protein as a CBP501-binding site.
    Matsumoto Y, Shindo Y, Takakusagi Y, et al. · Bioorg Med Chem · 2012
  8. Randomized phase II trial of pemetrexed/cisplatin with or without CBP501 in patients with advanced malignant pleural mesothelioma.
    Krug LM, Wozniak AJ, Kindler HL, et al. · Lung Cancer · 2015
    PubMed: PMID 25047675 · NCT04953962 · Pancreatic Neoplasms

Publications by year

20002024: 8 publications.

2000
1
2007
1
2011
1
2012
2
2015
2
2024
1

Publications by indication

Pancreatic Neoplasms (3)

  • A multicenter, randomized phase 2 study to establish combinations of CBP501, cisplatin and nivolumab for ≥3rd-line treatment of patients with advanced pancreatic adenocarcinoma.
    Eur J Cancer · 2024 · PMID 38422585 · NCT04953962
  • Randomized phase II trial of pemetrexed/cisplatin with or without CBP501 in patients with advanced malignant pleural mesothelioma.
    Lung Cancer · 2015 · PMID 25047675 · NCT04953962
  • CBP501-calmodulin binding contributes to sensitizing tumor cells to cisplatin and bleomycin.
    Mol Cancer Ther · 2012 · PMID 21831962 · NCT04953962

Neoplasms (2)

  • Screening of a library of T7 phage-displayed peptides identifies alphaC helix in 14-3-3 protein as a CBP501-binding site.
    Bioorg Med Chem · 2012 · PMID 22032894 · NCT00551512
  • Phase I studies of CBP501, a G2 checkpoint abrogator, as monotherapy and in combination with cisplatin in patients with advanced solid tumors.
    Clin Cancer Res · 2011 · PMID 21220472 · NCT00551512

Mesothelioma, Malignant (1)

  • Sensitization of cancer cells to DNA damage-induced cell death by specific cell cycle G2 checkpoint abrogation.
    Cancer Res · 2000 · PMID 10606229 · NCT00700336

Lung Neoplasms (1)

  • Cell cycle phenotype-based optimization of G2-abrogating peptides yields CBP501 with a unique mechanism of action at the G2 checkpoint.
    Mol Cancer Ther · 2007 · PMID 17237275 · NCT00942825

Publications by journal

All CBP501 publications (8)

2024 (1 paper)

  1. A multicenter, randomized phase 2 study to establish combinations of CBP501, cisplatin and nivolumab for ≥3rd-line treatment of patients with advanced pancreatic adenocarcinoma.
    Enzler T, Nguyen A, Misleh J, et al. · Eur J Cancer · 2024 · Derived
    PubMed: PMID 38422585 · NCT04953962 · Pancreatic Neoplasms

2015 (2 papers)

  1. Activation of Nrf2 pathways correlates with resistance of NSCLC cell lines to CBP501 in vitro.
    Mine N, Yamamoto S, Kufe DW, et al. · Mol Cancer Ther · 2015 · Background
  2. Randomized phase II trial of pemetrexed/cisplatin with or without CBP501 in patients with advanced malignant pleural mesothelioma.
    Krug LM, Wozniak AJ, Kindler HL, et al. · Lung Cancer · 2015 · Background
    PubMed: PMID 25047675 · NCT04953962 · Pancreatic Neoplasms

2012 (2 papers)

  1. CBP501-calmodulin binding contributes to sensitizing tumor cells to cisplatin and bleomycin.
    Mine N, Yamamoto S, Saito N, et al. · Mol Cancer Ther · 2012 · Background
    PubMed: PMID 21831962 · NCT04953962 · Pancreatic Neoplasms
  2. Screening of a library of T7 phage-displayed peptides identifies alphaC helix in 14-3-3 protein as a CBP501-binding site.
    Matsumoto Y, Shindo Y, Takakusagi Y, et al. · Bioorg Med Chem · 2012 · Background

2011 (1 paper)

  1. Phase I studies of CBP501, a G2 checkpoint abrogator, as monotherapy and in combination with cisplatin in patients with advanced solid tumors.
    Shapiro GI, Tibes R, Gordon MS, et al. · Clin Cancer Res · 2011 · Trial result

2007 (1 paper)

  1. Cell cycle phenotype-based optimization of G2-abrogating peptides yields CBP501 with a unique mechanism of action at the G2 checkpoint.
    Sha SK, Sato T, Kobayashi H, et al. · Mol Cancer Ther · 2007 · Background

2000 (1 paper)

  1. Sensitization of cancer cells to DNA damage-induced cell death by specific cell cycle G2 checkpoint abrogation.
    Suganuma M, Kawabe T, Hori H, et al. · Cancer Res · 2000 · Background
    PubMed: PMID 10606229 · NCT00700336 · Mesothelioma, Malignant

Sources and methodology

This page summarizes published evidence for general reference and does not constitute medical advice. For clinical decisions, consult the linked primary publications and your healthcare provider. Data sourced from PubMed and the ClinicalTrials.gov / AACT database maintained by the Clinical Trials Transformation Initiative (CTTI).